December 18, 2016
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Emotional Acting With Lyme: Pretending To Feel A Certain Way During A Social Interaction Based On Your Memory
November 30, 2016
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Samento and lyme disease treatment
An herb with powerful immune system modulating properties
Samento is produced from the inner bark or roots of the vine plant Uncaria Tomentosa, which is found in the Peruvian Rain Forest. Its hook-like thorns give its more commonly known tetracyclic oxindole alkaloid possessing brother, Cat's Claw, its name.
Samento and Lyme Disease
Samento is most commonly used for its ability to modulate the immune system by increasing the activity of white blood cells. White bloods cells are vital when it comes to protecting the body from foreign invaders such as bacteria and viruses. They're also responsible for generating antibodies which are used by the Western Blot Test for diagnosing Lyme Disease.
Samento also contains powerful antibacterial, antiviral, antifungal, and anti-inflammatory properties, which are all beneficial to a person battling Lyme Disease.
Samento is available as a tincture or in capsule form. According to Nutramedix, 3 drops of the Samento tincture has approximately the same clinical effect as one 600mg capsule of Samento. There are 600 drops in the Samento tincture. It should be noted that if you're taking the prescription drug Flagyl, you should not ingest it with alcohol. Samento in tincture form is diluted with alcohol.
Samento vs Cat's Claw
Cat's Claw contains the chemotypes tetracyclic oxindole alkaloids, or TOA, and pentacyclic oxindole alkaloids, or POA. TOA disrupts the function of the nervous system and also inhibits the effects of POA, the chemotype responsible for modulating the immune system.
Samento is in fact Cat's Claw, but it is a rare form of the plant deficient of TOA. Samento contains only pentacyclic oxindole alkaloids, or POA, which can modulate the immune system without the inhibiting affects of TOA. Samento is said to be many times more powerful than Cat's Claw as an antibiotic and immune system modulator because of its deficiency of the chemotype TOA, which is a POA inhibitor.